Cockles warmed: E. coli O157 strikes UK child, school says wasn’t us

Parents have been warned to be alert for signs of a “very infectious” strain of E. coli after a pupil from Thurnby Lodge Primary, in Leicester, contracted the O157 strain of the bug.

The source of the infection, caught over the half-term holidays, is not known, the school said. The severity of the illness has not been disclosed, but pupils in the same
class were sent letters by Public Heath England.

A spokeswoman for the school said, in her best British bureaucratese, “Thurnby Lodge Primary has received no further notifications of such incidents and it is deemed likely that the child was infected outside of school during the holidays. The letters from Public Health England came via the academy and were just a precautionary measure.”

The cockles of the parents were warmed.

And what kind of school is named, Thumby?

Handwashing is never enough: Texas family says sons infected with E. coli at petting zoo

An Azle family wants to warn others after both their young boys were hospitalized with E. coli earlier this year.

“It’s awful. You can’t do anything but just sit there and watch your child hurt,” Emily Miller told WFAA.

Miller’s sons Brayden, 7, and Dylan, 5, were both diagnosed with an E. coli infection, and Dylan’s case impacted his kidneys. Miller said he required dialysis, and he was hospitalized for 27 days, including several nights in the ICU.

“It’s such a crazy thought that this could happen,” Miller said.

She was surprised by the intensity of the illness, but also by where her boys may have come into contact with E. coli. She said doctors believe they were likely contaminated while the family was visiting a petting zoo.

“I wasn’t aware that you could get it from animals and livestock,” Miller said.

She took the boys to the petting zoo back in January, and four days later her oldest was in the hospital.

Both brothers are now doing well, though Dylan is still on blood pressure medicine due to the illness, Miller said.

The Centers for Disease Control says petting zoos do pose risks, as livestock can carry E. coli bacteria. The CDC’s advice is to wash hands with soap and water immediately after being near animals, whether you touch them or not.

The CDC also says that soap and water is more effective than instant hand sanitizers, and if sanitizers are the only option, go ahead and use them but follow up with soap and water as soon as possible.

A table of petting zoo outbreaks is available at http://barfblog.com/wp-content/uploads/2014/04/Petting-Zoo-Outbreaks-Table-4-8-14.xlsx.

Erdozain G, Kukanich K, Chapman B, Powell D. 2012. Observation of public health risk behaviours, risk communication and hand hygiene at Kansas and Missouri petting zoos – 2010-2011. Zoonoses Public Health. 2012 Jul 30. doi: 10.1111/j.1863-2378.2012.01531.x. [Epub ahead of print]

Abstract below:

Observation of public health risk behaviors, risk communication and hand hygiene at Kansas and Missouri petting zoos – 2010-2011Outbreaks of human illness have been linked to visiting settings with animal contact throughout developed countries. This paper details an observational study of hand hygiene tool availability and recommendations; frequency of risky behavior; and, handwashing attempts by visitors in Kansas (9) and Missouri (4), U.S., petting zoos. Handwashing signs and hand hygiene stations were available at the exit of animal-contact areas in 10/13 and 8/13 petting zoos respectively. Risky behaviors were observed being performed at all petting zoos by at least one visitor. Frequently observed behaviors were: children (10/13 petting zoos) and adults (9/13 petting zoos) touching hands to face within animal-contact areas; animals licking children’s and adults’ hands (7/13 and 4/13 petting zoos, respectively); and children and adults drinking within animal-contact areas (5/13 petting zoos each). Of 574 visitors observed for hand hygiene when exiting animal-contact areas, 37% (n=214) of individuals attempted some type of hand hygiene, with male adults, female adults, and children attempting at similar rates (32%, 40%, and 37% respectively). Visitors were 4.8x more likely to wash their hands when a staff member was present within or at the exit to the animal-contact area (136/231, 59%) than when no staff member was present (78/343, 23%; p<0.001, OR=4.863, 95% C.I.=3.380-6.998). Visitors at zoos with a fence as a partial barrier to human-animal contact were 2.3x more likely to wash their hands (188/460, 40.9%) than visitors allowed to enter the animals’ yard for contact (26/114, 22.8%; p<0.001, OR= 2.339, 95% CI= 1.454-3.763). Inconsistencies existed in tool availability, signage, and supervision of animal-contact. Risk communication was poor, with few petting zoos outlining risks associated with animal-contact, or providing recommendations for precautions to be taken to reduce these risks.

Best practices for planning events encouraging human-animal interactions

Zoonoses and Public Health 62:90-99, 2015

G. Erdozain , K. KuKanich , B. Chapman  and D. Powell

http://onlinelibrary.wiley.com/doi/10.1111/zph.12117/abstract?deniedAccess

Educational events encouraging human–animal interaction include the risk of zoonotic disease transmission. It is estimated that 14% of all disease in the US caused by Campylobacter spp., Cryptosporidium spp., Shiga toxin-producing Escherichia coli (STEC) O157, non-O157 STECs, Listeria monocytogenes, nontyphoidal Salmonella enterica and Yersinia enterocolitica were attributable to animal contact. This article reviews best practices for organizing events where human–animal interactions are encouraged, with the objective of lowering the risk of zoonotic disease transmission.

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Color sucks: Use a thermometer and stick it in for food safety

safefood Ireland has joined the UK Food Standards Agency in providing terrible advice about how to cook burgers.

A recipe for summer beef burgers (may a fine solstice greet our Northern and Southern friends) endorsed by safefood says:

“Before serving, ensure that the burgers are cooked thoroughly. Cut into them with a clean knife and check that they are piping hot all the way through, there is no pink meat remaining and that the juices run clear.”

Meanwhile, FSA issued a Safe Summer Food guide as UK picnickers head out in the sun (there’s sun in the UK?). The guidelines were in part based results of a self-reported survey, which is largely meaningless but something FSA likes to do.

The Morning Advertiser has more details on the hoops FSA seems willing to jump through to ensure the safety of rare burgers including:

  • sourcing the meat only from establishments which have specific controls in place to minimise the risk of contamination of meat intended to be eaten raw or lightly cooked;
  • ensuring that the supplier carries out appropriate testing of raw meat to check that their procedures for minimising contamination are working;
  • Strict temperature control to prevent growth of any bugs and appropriate preparation and cooking procedures;
  • notifying their local authority that burgers that aren’t thoroughly cooked are being served by the business; and,
  • providing advice to consumers, for example on menus, regarding the additional risk.

The advice from these self-proclaimed science-based agencies is at odds with, uh, science.

It has been known for over two decades that color is a lousy indicator of safety in hamburger.

The latest addition to this work comes from Djimsa et al. in the Dept. of Animal Science at Oklahoma State Univ., who wrote in the Journal of Food Science earlier this year that:

Premature browning is a condition wherein ground beef exhibits a well-done appearance before reaching the USDA recommended internal cooked meat temperature of 71.1 °C; however, the mechanism is unclear.

The objectives of this study were: (1) to determine the effects of packaging and temperature on metmyoglobin reducing activity (MRA) of cooked ground beef patties and (2) to assess the effects of temperature and pH on thermal stability of NADH-dependent reductase, lactate dehydrogenase (LDH), and oxymyoglobin (OxyMb) in-vitro.

Beef patties (lean: fat = 85:15) were packaged in high-oxygen modified atmosphere (HiOX-MAP) or vacuum (VP) and cooked to either 65 or 71 °C. Internal meat color and MRA of both raw and cooked patties were determined. Purified NADH-dependent reductase and LDH were used to determine the effects of pH and temperature on enzyme activity. MRA of cooked patties was temperature and packaging dependent (P < 0.05). Vacuum packaged patties cooked to 71 °C had greater (P < 0.05) MRA than HiOX-MAP counterparts.

Thermal stability of OxyMb, NADH-dependent reductase, and LDH were different and pH-dependent. LDH was able to generate NADH at 84 °C; whereas NADH-dependent reductase was least stable to heat.

The results suggest that patties have MRA at cooking temperatures, which can influence cooked meat color.

Effects of metmyoglobin reducing activity and thermal stability of NADH-dependent reductase and lactate dehydrogenase on premature browning in ground beef

Journal of Food Science, 2017 Feb, 82(2):304-313, doi: 10.1111/1750-3841.13606. Epub 2017 Jan 18.

https://www.ncbi.nlm.nih.gov/pubmed/28099768

French boy permanently disabled by E. coli in frozen beef

In June, 2011, eight children in Northern France were initially diagnosed with E. coli O157 after eating beef burgers bought from German discount retailer, Lidl.

In May 2012, the Institut de veille sanitaire summarized the outbreak, and revealed 17 children were sickened, 16 from E. coli O157-O177 and 1 due to E. coli O157-O26.

Now, a trial has begun for two former executives of French frozen food company SEB, charged with a “deliberate violation of safety obligations” that put customers at risk and caused involuntary injuries. Their trial began on Tuesday, June 6, and the two men face prison if convicted.

SEB has since gone out of business.

According to The Local, in 2011 a two-year-old boy named Nolan Moittie was one of 17 people in France who became seriously ill after eating steak hachés, or chopped steak patties, that were contaminated with E. coli bacteria, and which had been sold frozen at a Lidl grocery store. The illness caused the two-year-old boy to have a heart attack and fall into a coma while in the hospital.

The E. coli infection caused irreversible damage, and while Moittie survived and is now eight years old, he can’t talk and no longer has the use of 80 percent of his body. Doctors say the damage is irreversible.

But neither man is accepting responsibility and the defense is claiming that the illness from the minced beef was a result of consumers not storing and preparing them properly. 
 
Just cook it doesn’t cut it.
“Money as they say, won’t bring you happiness,  and it won’t help my son get back to how he was before,” his mother Priscilla said.
 
Steak hachés are a staple dish in France, particularly among children. In 2009 some 250,000 tonnes were sold, half of which were sold as frozen products.

1 dead, 29 sick from E. coli O157 linked to minced meat in Germany

We report an ongoing, protracted and geographically dispersed outbreak of hemolytic uremic syndrome (HUS) and gastroenteritis in Germany, involving 30 cases since December 2016. The outbreak was caused by the sorbitol-fermenting immotile variant of Shiga toxin-producing (STEC) Escherichia coli O157.

Molecular typing revealed close relatedness between isolates from 14 cases. One HUS patient died. Results of a case–control study suggest packaged minced meat as the most likely food vehicle. Food safety investigations are ongoing.

Ongoing hemolytic uremic syndrome (HUS) outbreak caused by sorbitol-fermenting (SF) shiga toxin-producing Escherichia coli (STEC) O157, Germany, December 2016 to May 2017

Eurosurveillance, vol. 22, issue 21, 25 May 2017, S Vygen-Bonnet, B Rosner, H Wilking, A Fruth, R Prager, A Kossow, C Lang, S Simon, J Seidel, M Faber, A Schielke, K Michaelis, A Holzer, R Kamphausen, D Kalhöfer,S Thole , A Mellmann, A Flieger, K Stark

http://www.eurosurveillance.org/ViewArticle.aspx?ArticleId=22805

 

We’ve learned lessons but we’re not telling anyone: Public Health England accused of E. coli O55 cover up

In Dec. 2014, an outbreak of E. coli 055 was identified in Dorset, U.K. with at least 31 sickened. Public Health England (PHE) and local environmental health officials investigated and found nothing, other than cats were also being affected.

Tara Russell of the Bournemouth Echo reports a nurse who fears her family’s lives will never be the same again after contracting the deadly E. coli bug has accused health officials of a ‘cover up.’

Jessica Archer and her nephew Isaac Mortlock, now five, were among the first of 31 people in Dorset who battled for their lives when they were diagnosed with hemolytic uremic syndrome (HUS) as a result of E. coli O55 in summer 2014.

Three years on, Isaac suffers with severe seizures, must be peg fed for 10 hours each night and will need a kidney transplant and Jessica endures crippling head pains, fatigue and depression as a result of the bug.

But though the families’ lives have changed irreversibly, they feel let down by the Public Health England (PHE) investigation.

Jessica, who completed the London Marathon to raise awareness of her family’s plight, said: “This illness has robbed us. We are no longer the same people. It’s very frightening how life can suddenly change in an instant and I’m sure if all the other families were sat around the table they would say exactly the same.

“If someone attempts to murder someone, that is taken very seriously. We have been close to death through whatever reason that may be, yet we feel it was not taken at all seriously and more and more and more people suffered.

“It feels like a cover up and we just believe there are too many questions that have been left unanswered.”

Isaac and Jessica became ill after the family had eaten together at a restaurant. Medics originally put symptoms down to gastroenteritis but they were later diagnosed with a severe life-threatening complication which attacked their kidneys, liver and brain and fought for life. Today Jessica said they face a daily battle for recovery.

“We were discharged within a day of each other and we were so naïve. We thought we’d get our lives back together. How wrong were we.

“I used to be fit and active, now there’s not a day I can say ‘I feel well today.’”

Isaac today suffers severe health and behavioural problems.

The investigation closed in March 2016 without the affected families being made aware and failed to find the source. The outbreak was only confirmed by PHE in response to enquiries made by the Bournemouth Echo in November 2014 after it struck a children’s nursery in Blandford – months after the initial outbreak.

Jessica said: “We’ve been in the dark throughout with absolutely no communication about the outbreak, investigation or what has happened since. If they’d have thought we were important enough to find the cause, little babies and children may not have been put through the same hell.

“All we can hope for is that lessons have been learned so no other family ever has to go through the same horrendous ordeal we are living. For us, every morning is a constant reminder that life for us will never be the same.”

This is normal in the U.K. where science-based agencies recommend cooking food to the standard of piping hot, and where 252 people were sickened with E. coli O157 in 2010 – 80 hospitalized, one death, possibly linked to potatoes and leeks – and the Food Standards Agency reminded people to wash their produce.

This is some fucked up shit.

Maybe that’s why I like John Oliver so much.

He says he’s British and has no human emotion.

It’s buried way, way down.

Russell of the Bournemouth Echo writes public health officials carried out a review of the E. coli outbreak in Dorset to ‘identify lessons learnt.’

Public Health England said it is a ‘learning organisation’ and ‘reflects on outbreaks’ however refused to reveal what these lessons were.

Dr Sarah Harrison, consultant in health protection at Public Health England South West said: “Our colleagues in Public Health England worked closely with partners to try and identify a possible common source of infection, but the investigations did not identify a single common source. It is very good news that there have been no further cases of infection with this strain in Dorset since the end of the outbreak in 2015, however we remain vigilant.

“PHE is a learning organisation and reflects on outbreaks to identify lessons learnt and to continually improve our response. A review of this outbreak was conducted at the time by staff involved in line with standard procedures.

“E coli VTEC can be a very serious infection and can be passed easily from person to person and young children are particularly easily affected. We know that the bacteria causing the infection can survive in the environment, so good hand hygiene is important to prevent the spread. Wash hands thoroughly using soap and water after using the toilet, before and after handling food and after contact with animals including farm animals. Small children should be supervised in washing their hands. Remove any loose soil before storing vegetables and thoroughly wash all vegetables and fruit that will be eaten raw.”

PHE said the investigation at the time was extensive with involvement from many organisations.

A statement read: “Control measures included extended screening and exclusion of cases and high risk contacts. Public Health England and the Animal and Plant Health Agency put in place enhanced surveillance of faecal samples in Dorset laboratories and environmental sampling to help determine the extent of this organism in the community. The only link common to all the cases was that they either lived in or had close links to the county. The outbreak investigation closed in March 2016.”

As John Oliver would say, cool.

As Jessica Archer would say, fuck off you bureaucratic assholes who spend work time watching goats singing Taylor Swift songs on the the Intertubes.

Bambi poops in water, 4 kids get sick with E. coli O157, 2016

In May 2016, an outbreak of Shiga toxin–producing Escherichia coli O157 infections occurred among children who had played in a stream flowing through a park. Analysis of E. coli isolates from the patients, stream water, and deer and coyote scat showed that feces from deer were the most likely source of contamination.

In the United States, recreational water is a relatively uncommon source of Shiga toxin–producing Escherichia coli (STEC) O157 outbreaks (1). We describe an outbreak of STEC O157 infections among children exposed to a contaminated stream in northern California, USA, and provide laboratory evidence establishing wildlife as the source of water contamination.

In May 2016, four cases of Shiga toxin (Stx) 1– and 2–producing E. coli O157 infection were reported to a local health department in northern California; investigation revealed a common source of exposure. The case-patients, ranging in age from 1 to 3 years, had played in a stream adjacent to a children’s playground within a city park. Exposure of the case-patients to the stream occurred on 3 separate days spanning a 2-week period. Two case-patients are known to have ingested water while playing in the stream. Two case-patients were siblings. All case-patients had diarrhea and abdominal cramps; bloody diarrhea was reported for 3. One case-patient was hospitalized with hemolytic uremic syndrome.

The stream is a second-order waterway located in a northern California community of ≈7,500 residents. At the time of exposures, stream flow was <30 ft3/s. The land upstream is not used for agricultural activities such as livestock production. The community is serviced by a public sewer system; inspection of sewer lines indicated no breach to the system.

Water samples were collected from the exposure site 7 days after the last case-patient was exposed and weekly thereafter for 17 weeks; samples were tested quantitatively for fecal indicator organisms. Throughout the study period, all water samples exceeded recreational water quality limits for E. coli and enterococci levels (2). Water samples were also cultured for STEC isolation and PCR detection of stx1 and stx2 (3). Stx1- and Stx2-producing E. coli O157 were isolated from stream water each week for the first 4 weeks. Additionally, an Stx2-producing E. coli non-O157 strain was isolated from the stream in the first week of sampling. Enrichment broth cultures of water samples were also positive by PCR for stx1 and stx2 for the first 4 weeks of sampling. Thereafter, both stx1 and stx2, or stx2 only, were intermittently detected in enrichment broth cultures for 9 additional weeks.

In the absence of an obvious source (e.g., upstream agricultural operation or sewer leak), wildlife was considered as a possible contributor to water contamination. Thirteen fresh wildlife scat specimens were collected along the stream for STEC culture and PCR. Of the 13 scat specimens, 8 originated from deer, 2 from raccoon, and 1 each from coyote, turkey, and river otter. Six scat specimens (4 deer, 1 coyote, 1 river otter) were positive for stx1 and stx2 or for stx2 by PCR (Technical Appendix[PDF – 16 KB – 1 page]). Stx1- and Stx2-producing E. coli O157 were isolated from deer scat and coyote scat. An Stx2-producing E. coli non-O157 strain was isolated from a deer scat specimen. The animal origin of the coyote and river otter scat specimens were definitively identified by partial DNA sequencing of mitochondrial cytochrome b (4).

To assess strain relatedness, we compared STEC O157 isolates from the case-patients, water, deer scat, and coyote scat by using pulsed-field gel electrophoresis (PFGE) and multilocus variable-number tandem-repeat analysis (MLVA) (5). PFGE patterns for XbaI-digested genomic DNA were highly similar among all isolates; only slight variations were found in the lower-sized bands (Figure). PFGE patterns for genomic DNA samples digested with BlnI also demonstrated a high degree of similarity (data not shown). Furthermore, MLVA profiles were identical for the case-patient, water, and deer scat isolates; only the coyote scat isolate differed from the main profile by 2 repeats at a single locus (VNTR_3).

This study provides laboratory evidence linking STEC O157 infections with the ingestion of recreational water that was probably contaminated by wildlife scat. Wild ruminants, including deer and elk, are known carriers of STEC and have been connected to outbreaks of human infections (69). We detected STEC in 50% of deer scat specimens collected from the stream bank. One of these specimens, found 1.5 miles upstream of the exposure site, contained an E. coli O157 isolate that was highly similar by molecular subtyping to case-patient and water isolates. These findings support the likelihood that feces from deer carrying STEC were the source of water contamination or, at the very least, contributed to the persistence of STEC in the water. It is unknown whether the STEC detected in coyote and river otter scat represents carriage or transitory colonization within these animals.

The common risk factor among the case-patients in this STEC O157 outbreak was exposure to a natural stream within a city park. After the outbreak was recognized, signs warning of bacterial contamination were posted along the stream. No further STEC O157 infections attributed to stream water exposure were reported.

Dr. Probert is the assistant director for the Napa-Solano-Yolo-Marin County Public Health Laboratory. His research interests focus on the development of molecular diagnostic tools for the detection of infectious agents.

Acknowledgment

We thank Frank Reyes, Keith Snipes, and Nailah Souder for their technical assistance; the County of Marin Health and Human Services and Environmental Health Services for information about the epidemiologic and environmental investigation; and the Microbial Diseases Laboratory Branch of the California Department of Public Health and the Santa Clara County Public Health Laboratory for the molecular subtyping data.

References

Heiman KE, Mody RK, Johnson SD, Griffin PM, Gould LH. Escherichia coli O157 outbreaks in the United States, 2003–2012. Emerg Infect Dis. 2015;21:1293–301. DOIPubMed

United States Environmental Protection Agency. 2012. Recreational water quality criteria. Office of Water 820-F-12–058 [cited 2017 Apr 13]. https://www.epa.gov/sites/production/files/2015-10/documents/rwqc2012.pdf

Probert WS, McQuaid C, Schrader K. Isolation and identification of an Enterobacter cloacae strain producing a novel subtype of Shiga toxin type 1. J Clin Microbiol. 2014;52:2346–51. DOIPubMed

Parson W, Pegoraro K, Niederstätter H, Föger M, Steinlechner M. Species identification by means of the cytochrome b gene. Int J Legal Med. 2000;114:23–8. DOIPubMed

Hyytia-Trees E, Lafon P, Vauterin P, Ribot EM. Multilaboratory validation study of standardized multiple-locus variable-number tandem repeat analysis protocol for Shiga toxin–producing Escherichia coli O157: a novel approach to normalize fragment size data between capillary electrophoresis platforms. Foodborne Pathog Dis. 2010;7:129–36. DOIPubMed

Fischer JR, Zhao T, Doyle MP, Goldberg MR, Brown CA, Sewell CT, et al. Experimental and field studies of Escherichia coli O157:H7 in white-tailed deer. Appl Environ Microbiol. 2001;67:1218–24. DOIPubMed

Keene WE, Sazie E, Kok J, Rice DH, Hancock DD, Balan VK, et al. An outbreak of Escherichia coli O157:H7 infections traced to jerky made from deer meat. JAMA. 1997;277:1229–31. DOIPubMed

Rounds JM, Rigdon CE, Muhl LJ, Forstner M, Danzeisen GT, Koziol BS, et al. Non-O157 Shiga toxin–producing Escherichia coli associated with venison. Emerg Infect Dis. 2012;18:279–82. DOIPubMed

Laidler MR, Tourdjman M, Buser GL, Hostetler T, Repp KK, Leman R, et al. Escherichia coli O157:H7 infections associated with consumption of locally grown strawberries contaminated by deer. Clin Infect Dis. 2013;57:1129–34. DOIPubMed

 Contaminated stream water as source for Escherichia coli O157 illness in children

05.may.17

William S. Probert, Glen M. Miller, and Katya E. Ledin

Emerging Infectious Diseases, vol. 23, no. 7, July 2017

https://wwwnc.cdc.gov/eid/article/23/7/17-0226_article

840X greater risk from raw milk and products

Risk comparisons are generally risky.

I avoid them.

But if some folks are going to push a point, expect some push back.

Risk comparisons depend on meals consumed. Not many Americans consume raw milk or raw milk cheese, yet the products are continuously the source of outbreaks.

The following abstract of a paper takes a stab at quantifying the per-meal problem.

Why has no one published about the imagined safety of raw milk products in a scientific journal?

Because it’s another food safety fairytale.

Until credible data is presented, all the naturalist wankers can take the advice of novelist Kurt Vonnegut, “Why don’t you take a flying fuck at a rolling doughnut? Why don’t you take a flying fuck at the mooooooooooooon?”

And stop wasting public health resources, assholes.

Outbreak-related disease burden associated with consumption of unpasteurized cow’s milk and cheese, United States, 2009-2014

Emerging Infectious Diseases, vol. 23, no. 6, June 2017, Solenne Costard , Luis Espejo, Huybert Groenendaal, and Francisco J. Zagmutt

https://wwwnc.cdc.gov/eid/article/23/6/15-1603_article
The growing popularity of unpasteurized milk in the United States raises public health concerns. We estimated outbreak-related illnesses and hospitalizations caused by the consumption of cow’s milk and cheese contaminated with Shiga toxin–producing Escherichia coli, Salmonella spp., Listeria monocytogenes, and Campylobacter spp. using a model relying on publicly available outbreak data. In the United States, outbreaks associated with dairy consumption cause, on average, 760 illnesses/year and 22 hospitalizations/year, mostly from Salmonella spp. and Campylobacter spp.

Unpasteurized milk, consumed by only 3.2% of the population, and cheese, consumed by only 1.6% of the population, caused 96% of illnesses caused by contaminated dairy products. Unpasteurized dairy products thus cause 840 (95% CrI 611–1,158) times more illnesses and 45 (95% CrI 34–59) times more hospitalizations than pasteurized products. As consumption of unpasteurized dairy products grows, illnesses will increase steadily; a doubling in the consumption of unpasteurized milk or cheese could increase outbreak-related illnesses by 96%.

People are getting sick E. coli O157 outbreak at Boston’s Chicken & Rice Guys

Megan Woolhouse of the Boston Globe reports an E. coli O157 outbreak shuttered three locations of the Chicken & Rice Guys, as well as its fleet of Middle Eastern food trucks, Boston health inspectors said Tuesday.

The department confirmed seven cases of E. coli stemming from the Chicken & Rice Guys Allston location, which supplies food to the chain’s other outposts. The problems led to the temporary suspension of its operating license, Boston Inspectional Services Commissioner William Christopher Jr. said.

“We’re taking this very seriously,” Christopher said. “People are getting sick.”

He added that he did not know the condition of any of the people who were affected.

The company’s four food trucks, which rotate locations around Greater Boston, were taken off the road Tuesday afternoon, said Phanna Ky, general manager of the chain’s Medford restaurant, the only location that remained open Tuesday evening.

Christopher said Boston does not have jurisdiction over the Medford location.

Chicken & Rice Guys officials could not be reached.

According to Boston Inspectional Services, the city received an anonymous complaint and opened an investigation Tuesday. Public health officials remained at the Allston site throughout the afternoon trying to determine a specific source of the outbreak, Christopher said.

He added that the department will meet with the chain’s owner on Wednesday morning to discuss a course of action.

Steak tartare: A special kind of stupid

A favorite line in the ice hockey linesman course I take every year to be recertified is, “that player exhibited a special kind of stupid”

Cooks and purveyors of food porn exhibit their own special kind of stupid, especially around raw beef.

The N.Y. Times continues its long history of bad food porn-based advice because, they’re New Yorkers, and they are their own special kind of stupid: at least the uppity ones.

Gabrielle Hamilton writes in the New York Times Cooking section that a hand-chopped mound of cold raw beef, seasoned perfectly, at around 3 o’clock in the afternoon on New Year’s Day, with a cold glass of the hair of the Champagne dog that bit you the night before, will make a new man out of you.

Hamilton writes the recipe calls for 8-10 ounces highest-quality beef tenderloin … and to nestle each yolk, still in its half shell if using raw, into the mound, and let each guest turn the yolk out onto the tartare before eating.

Nary a mention of Shiga-toxin producing E. coli or Salmonella or Campylobacter.